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71. Haber SN, Fudge JL, McFarland NR. Striatonigrostriatal pathways in primates form an ascending spiral from the shell to the dorsolateral striatum. J Neurosci 2000;20:2369–2382. 72. Parent A, Sato F, Wu Y, et al. Organization of the basal ganglia: the importance of axonal collateralization. Trends Neurosci. 2000;23:S20–S27. 73. Parent M, Lévesque M, Parent A. The pallidofugal projection system in primates: evidence for neurons branching ipsilaterally and contralaterally to the thalamus and brainstem.
An autoradiographic analysis of the efferent connections from premotor and adjacent prefrontal regions (areas 6 and 9) in macaca f ascicularis. Brain Behav Evol 1978;15:185–234. 108. Watson RT, Valenstein E, Heilman KM. Thalamic neglect. Possible role of the medial thalamus and nucleus reticularis in behavior. Arch Neurol 1981;38:501–506. 109. Matsumoto N, Minamimoto T, Graybiel AM, et al. Neurons in the thalamic CM-Pf complex supply striatal neurons with information about beha viorally significant sensory events.
This syndrome has been reported with focal lesions in volving either the globus pallidus after anoxia (12) or bilateral hemorrhages (24) or the caudate nucleus after vascular accidents (ischemia, hematoma, or lacunae) (16,20). The relation between autoacti vation deficits and the striatopallidal comple x has been confirmed by a meta-analysis of beha vioral changes following lesions of the basal ganglia: 6 of the 7 patients with pallidal lesions and 17 of the 33 patients with caudate lesions p resented behavioral inertia, but none of the patients Mental Emptiness The fact that patients with basal ganglia lesions can remain inactive for hours without complaining of boredom may be e xplained by a lack of spontaneous mental life.
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